IL-17-producing CD4 tissue-resident memory T cells mediate protective immunity against Bordetella pertussis infection in the nasal mucosae

Exploring the intricacies of host-pathogen communication is vital in order to understand why
some microbes persist within a host, while others are effectively cleared. As such,
host-pathogen communication is an urgent area of research underpinning human health
and disease, especially given the emerging concerns of antibiotic resistance.
Understanding host-pathogen pathways requires intricate knowledge in the fields of both
microbiology and immunology.

Living in a world of specialists, where scientists sometimes invest their whole career
studying one organism, a specific pathway or a protein, multidisciplinary discussions of
host-pathogen interactions are highly needed. This international conference will provide
such a forum for scientific exchange at the highest level, between scientists from different
research areas related to host-pathogen interactions, with the aim to trigger fruitful
collaborations addressing the urgent questions in the field.

This meeting has an additional special focus of bringing researchers from Ireland, Denmark
and Israel together to learn about cutting edge biology and form new collaborations.
Thus, the conference will be a showcase for the exciting and original research emanating
from these three small but innovative countries.

Host-Pathogen Communication (HPC) 2021

Mechanisms to limit Sars-CoV2 infection

A novel role of mouse PYHIN protein Ifi207 in transcriptional regulation of pro-inflammatory genes

Trypanosoma Brucei secreted aromatic ketoacids represent novel therapies for inflammatory diseases through suppression of pro-inflammatory responses in primary human immune cells.

A subset of CD4+ effector memory T cells suppress T cell-mediated immunity to pulmonary viral infection via integrin αvβ8-dependent activation of latent TGFβ

SLC7a5 amino acid transporter regulates metabolic responses in activated NK cells in vivo.

Enrichment of a novel, pathogenic macrophage population with distinct transcriptional and metabolic signatures in Rheumatoid Arthritis synovial tissue

Welcome to TBSI

Opening remarks