Background and Aim: Under the influence of asphyxia irreversible changes occur, including the failure of compensatory mechanisms, pathological acidosis and bone marrow irritation. The factors determining the formation of hemostasis in premature infants who have suffered asphyxia have not been sufficiently studied yet, therefore, further studies are required. 
The aim of our investigation is to study the peripheral blood picture in preterm infants with and without asphyxia, born from mothers with preeclampsia (PE) .  
Materials and Metods: All infants involved in this study were divided into the following two groups: 
The main group (from mothers with PE, GA= 27-36 weeks) that was also divided into subgroups: 1A - preterm infants with asphyxia (N=28), 1B - preterm infants without asphyxia (N-20); and the control group (N=30)
  
Results: Fe and transferrin levels were significantly lower in infants of 1A subgroup, than in infants of 1B subgroup.  On days 5-7 Fe level was 82.6±4.3 in A subgroup infants, and 92.0±5.4 in B subgroup newborns(p<0.001).
                                          
Conclusion: As we know, iron - deficiency anemia is not typical for preterm infant on their first days of life. 
It could be explained by the exhaustion of the compensatory capacity of the preterm newborn’s organism and the effects of hypoxia and immaturity in the background.

CONDITION OF ERYTHROPOIESIS IN PRETERM INFANTS, BORN IN ASPHYXIA FROM MOTHERS WITH PREECLAMPSIA