The peach-potato aphid, Myzus persicae, is a globally important crop pest that causes feeding damage and transmission of viral diseases on a variety of crops. It is normally controlled through the use of chemical insecticides as seed treatments and/or sprays, although strong selection pressure over many years has resulted in populations that are resistant to most of the major chemical classes. This talk will focus on the main target site mechanisms that have been identified in this species; modified acetylcholinesterase (conferring resistance to dimethylcarbamates), kdr-type mutations in the voltage-gated sodium channel (resistance to pyrethroids) and the more recent nicotinic acetylcholine receptor mutation (conferring resistance to neonicotinoids). For each mechanism, I will review how the characterisation of resistance and identification of the casual mutation(s) has led to an improved understanding of insecticide mode of action, target/insecticide interaction and target selectivity, as well as providing opportunities for the development of high throughput, PCR-based diagnostic assays for rapid monitoring of mutation frequencies in localised populations and options for resistance management.

Insecticide resistance in the peach potato aphid,<i> Myzus persicae</i>, Part 1: Mutations, modelling, and management

248th National Meeting (2014)

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Insecticide resistance in the peach potato aphid, Myzus persicae, Part 1: Mutations, modelling, and management